Lis1 is shown to situation microtubules within the progress spool [47], along with the man Lis1 homologue is vital pertaining to neuronal migration so when mutated brings about Lissencephaly, a serious neuronal migration defect seen as an effortless cerebral #links# surface, mind retardation along with seizures [48]. Now we have found out that Lis1 RNAi /GMR-Gal4 mutants get unusually created lami